25 Because a small number of component studies may limit the validity of the heterogeneity tests, we used a random- effects model, even when low heterogeneity was detected order priligy Immortalized mammary epithelial cell lines with normal phenotype including MCF10A 12A, S1, NMuMG, epH4, human mammary epithelial cell and HB2, breast cancer cell lines including MCF 7, T47D, MDA MB 231, T 42, SKBR 3, BT549 and primary mammary epithelial cells of both normal and tumour origin can be cultured and are commonly used to model the molecular contribution of MAPK signalling in normal epithelia and in breast cancer
25 Because a small number of component studies may limit the validity of the heterogeneity tests, we used a random- effects model, even when low heterogeneity was detected order priligy Immortalized mammary epithelial cell lines with normal phenotype including MCF10A 12A, S1, NMuMG, epH4, human mammary epithelial cell and HB2, breast cancer cell lines including MCF 7, T47D, MDA MB 231, T 42, SKBR 3, BT549 and primary mammary epithelial cells of both normal and tumour origin can be cultured and are commonly used to model the molecular contribution of MAPK signalling in normal epithelia and in breast cancer